Publication | Open Access
Colorectal cancer cells with the BRAFV600E mutation are addicted to the ERK1/2 pathway for growth factor-independent survival and repression of BIM
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Citations
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References
2008
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OncologyMedicineColorectal CancerGrowth Factor-independent SurvivalColorectal Cancer CellsTumor SuppressorCancer GeneticsRadiation OncologyCancer BiologyCell BiologyCell SignalingTumor BiologyBrafv600e Mutation
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