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Cellular Steady-State Levels of “High Risk” but Not “Low Risk” Human Papillomavirus (HPV) E6 Proteins Are Increased by Inhibition of Proteasome-Dependent Degradation Independent of Their p53- and E6AP-Binding Capabilities

Cellular Steady-state LevelsHuman Papillomavirus VaccinesE6ap-binding CapabilitiesPathologyProtein DegradationProteasome-dependent Degradation IndependentMedicineCell BiologyCancer ResearchTumor BiologyViral OncologyCancer-associated Virus