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Age-related cognitive deficits, impaired long-term potentiation and reduction in synaptic marker density in mice lacking the β-amyloid precursor protein

Synaptic PlasticityCognitive ScienceNeurodegenerative DiseasesAge-related Cognitive Deficitsβ-Amyloid Precursor ProteinLong-term PotentiationNeurochemical BiomarkersNeurologyNeuroscienceAging-associated DiseaseBrain PathologyMedicineSynaptic SignalingSocial Sciences