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Secreted amyloid β–protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

Molecular BiologySynaptic SignalingSocial SciencesPresenilin 1Alzheimer's DiseaseDegenerative PathologyProtein MisfoldingNeurologyBrain PathologyNeuropathologyMolecular NeuroscienceAmyloid β–ProteinNeurodegenerationProtective MechanismsNeurodegenerative DiseasesBiomarkersNeuroscienceSenile PlaquesMedicine