Kheem S. Bisht - Concepedia

Author

Kheem S. Bisht

Also Known As

K S Bisht, K. Bisht, K. S. Bisht, K.S Bisht, K.S. Bisht, Kanika Bisht, Kheem Bisht, Kheem S Bisht, Kheem S. Bisht, Kheem Singh Bisht

Publications

2.9K

Citations

H-Index

Concepts

All Affiliations

National Cancer Institute Center for Cancer Research National Institutes of Health Washington University in St. Louis University of Iowa

About

Kheem S. Bisht is an author at National Cancer Institute specializing in medicine, cell biology, and radiation oncology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	9	9	1.6K
2	Cell Biology	7	7	1.4K
3	Pharmacology	4	4	442
4	Natural Sciences	2	2	277
5	Molecular Biology	2	2	277

Kheem S. Bisht

Publications

2.8K

Citations

H-Index

Page 1

	Year	Citations
SIRT3 Is a Mitochondria-Localized Tumor Suppressor Required for Maintenance of Mitochondrial Integrity and Metabolism during Stress Hyunseok Kim, Krish Patel, Kristi Muldoon‐Jacobs, Cancer Cell Reductive StressTumor BiologyMitophagyMitochondrial FunctionMedicine	2010	791
<i>SIRT3</i> interacts with the <i>daf-16</i> homolog <i>FOXO3a</i> in the Mitochondria, as well as increases <i>FOXO3a</i> Dependent Gene expression Kristi Muldoon Jacobs, J. Daniel Pennington, Kheem S. Bisht, International Journal of Biological Sciences	2008	271
Simultaneous inhibition of hsp 90 and the proteasome promotes protein ubiquitination, causes endoplasmic reticulum-derived cytosolic vacuolization, and enhances antitumor activity Edward G. Mimnaugh, Wanping Xu, Michele D. Vos, Molecular Cancer Therapeutics	2004	210
Distinct effects on gene expression of chemical and genetic manipulation of the cancer epigenome revealed by a multimodality approach David Gius, Hengmi Cui, C. Matthew Bradbury, Cancer Cell ChromatinSystems BiologyMultimodality ApproachNatural SciencesEpigenetic Change	2004	182
Geldanamycin and 17-allylamino-17-demethoxygeldanamycin potentiate the in vitro and in vivo radiation response of cervical tumor cells via the heat shock protein 90-mediated intracellular signaling and cytotoxicity. Kheem S. Bisht, C. Matthew Bradbury, David Mattson, PubMed Heat Shock ProteinTumor Biology17-Allylamino-17-demethoxygeldanamycin PotentiateOncologyCervical Cancer	2003	178
Thioredoxin Reductase as a Potential Molecular Target for Anticancer Agents That Induce Oxidative Stress Dee Dee Smart, Karen Lynette Ortiz, David Mattson, Cancer Research	2004	118
Radiosensitizing and anti-proliferative effects of resveratrol in two human cervical tumor cell lines Imran Zoberi, C. Matthew Bradbury, Heather A. Curry, Cancer Letters OncologyCervical CancerRadiation TherapyAnti-proliferative EffectsMedicine	2002	108
A comparative study of radioprotection by Ocimum flavonoids and synthetic aminothiol protectors in the mouse. P U Devi, Kheem S. Bisht, M Vinitha British Journal of Radiology Body IrradiationMedicineRadiation EffectRadiation ExposureSynthetic Aminothiol Protectors	1998	104
BAT3 and SET1A Form a Complex with CTCFL/BORIS To Modulate H3K4 Histone Dimethylation and Gene Expression Phuongmai Nguyen, Gil Bar‐Sela, Lunching Sun, Molecular and Cellular Biology Histone ModificationsEpigenetic ChangeGeneticsMolecular BiologyEpigenetics	2008	95
Cytogenetic Studies in Human Blood Lymphocytes Exposed<i>In Vitro</i>to Radiofrequency Radiation at a Cellular Telephone Frequency (835.62 MHz, FDMA) William F. Pickard, Kheem S. Bisht, Belinda Z. Leal, Radiation Research	2001	86

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