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PI3Kδ drives the pathogenesis of experimental autoimmune encephalomyelitis by inhibiting effector T cell apoptosis and promoting Th17 differentiation

Autoimmune DiseaseAllergyT-regulatory CellImmunologyImmune RegulationCell DeathExperimental Autoimmune EncephalomyelitisImmunologic MechanismAutoimmunityImmunopathologyImmunotherapyMedicineTh17 Differentiation