Kelly Johnson‐Wood

Author

Kelly Johnson‐Wood

Also Known As

K Johnson-Wood, K. Johnson-Wood, K. Johnson‐Wood, K. L. Johnson‐Wood, K.L. Johnsonwood, KELLY JOHNSON‐WOOD, Kelly Johnson-Wood, Kelly Johnson-wood, Kelly Johnson‐Wood, Kelly L. Johnson-Wood

Publications

15.2K

Citations

H-Index

Concepts

All Affiliations

Athena Group (United States)Sunesis (United States)University of California San Diego Princeton University University of California, San Francisco

About

Kelly Johnson‐Wood is an author at Athena Group (United States) specializing in medicine, neurodegenerative diseases, and alzheimer's disease.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	16	14.4K
2	Social Sciences	5	5	6.6K
3	Health Sciences	4	4	2.5K
4	Natural Sciences	2	2	1.7K
5	Engineering	1	1	23

Kelly Johnson‐Wood

Publications

15.1K

Citations

H-Index

Page 1

	Year	Citations
Immunization with amyloid-β attenuates Alzheimer-disease-like pathology in the PDAPP mouse Dale Schenk, Robin Barbour, Whitney Dunn, Nature Neurodegenerative DiseasesAlzheimer's DiseaseImmunologyDegenerative PathologyProtein Misfolding	1999	3.1K
Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein Dora Games, David S. Adams, Ree Alessandrini, Nature Alzheimer-type NeuropathologyNeurodegenerative DiseasesAlzheimer's DiseaseMolecular NeuroscienceTransgenic Mouse Models	1995	2.5K
Peripherally administered antibodies against amyloid β-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease Frédérique Bard, Catherine Cannon, Robin Barbour, Nature Medicine Amyloid β-Peptide EnterNeurodegenerative DiseasesAlzheimer's DiseaseMedicineImmunology	2000	2K
High-Level Neuronal Expression of Aβ<sub>1–42</sub>in Wild-Type Human Amyloid Protein Precursor Transgenic Mice: Synaptotoxicity without Plaque Formation Lennart Mucke, Eliezer Masliah, Gui-Qiu Yu, Journal of Neuroscience Neurodegenerative DiseasesMolecular NeuroscienceAlzheimer's DiseaseAmyloid PlaquesDementia	2000	1.9K
Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid β-protein in both transfected cells and transgenic mice Martin Citron, David Westaway, Weiming Xia, Nature Medicine Neurodegenerative DiseasesAlzheimer's DiseaseMolecular NeuroscienceNatural SciencesDisease Increase Production	1997	1.3K
Functional gamma‐secretase inhibitors reduce beta‐amyloid peptide levels in brain Harry F. Dovey, Varghese John, J. P. Anderson, Journal of Neurochemistry Protein SecretionNeurochemical BiomarkersAmyloid Precursor ProteinSocial SciencesAlzheimer's Disease	2001	877
Reduction of β‐amyloid peptide<sub>42</sub> in the cerebrospinal fluid of patients with Alzheimer's disease Ruth Motter, Carmen Vigo‐Pelfrey, Dora Kholodenko, Annals of Neurology Apoe GenotypeNeurochemical BiomarkersCsf Aβ 42Social SciencesAlzheimer's Disease	1995	692
Amyloid precursor protein processing and Aβ <sub>42</sub> deposition in a transgenic mouse model of Alzheimer disease Kelly Johnson‐Wood, M. Lee, Ruth Motter, Proceedings of the National Academy of Sciences Transgenic Mouse ModelSocial SciencesAlzheimer's DiseaseDegenerative PathologyProtein Misfolding	1997	655
The secreted form of the Alzheimer's amyloid precursor protein with the Kunitz domain is protease nexin-II Tilman Oltersdorf, Lawrence C. Fritz, Dale B. Schenk, Nature Neurodegenerative DiseasesProtein SecretionAlzheimer's DiseaseMolecular NeuroscienceBiochemistry	1989	461
Amyloidogenic role of cytokine TGF-β1 in transgenic mice and in Alzheimer's disease Tony Wyss‐Coray, Eliezer Masliah, Margaret Mallory, Nature InflammationNeurodegenerative DiseasesAlzheimer's DiseaseAmyloidogenic RoleImmunology	1997	417

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