Markus Moser - Concepedia

Author

Markus Moser

Max Planck Institute of Biochemistry ORCID

Also Known As

L Plass, M Moser, M. MOSER, M. Moser, MARKUS MOSER, Markus MOSER, Markus Moser, Markus Moser, PhD, Max Moser, Michel Moser

284

Publications

19.5K

Citations

H-Index

204

Concepts

All Affiliations

Max Planck Institute of Biochemistry Ludwig-Maximilians-Universität München Max Planck Society University of Regensburg University of Bonn

About

Markus Moser is an author at Max Planck Institute of Biochemistry specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	42	67	9.3K
2	Natural Sciences	16	18	1.8K
3	Engineering	5	5	310
4	Health Sciences	4	4	287
5	Education	3	3	261

Markus Moser

164

Publications

17.5K

Citations

H-Index

Page 1

	Year	Citations
Prion protein (PrP) with amino-proximal deletions restoring susceptibility of PrP knockout mice to scrapie. Michael B. Fischer, Thomas Rülicke, Alex J. Raeber, The EMBO Journal Amino-proximal DeletionsCreutzfeldt-jakob DiseaseBiochemistryMedicineGenetics	1996	875
Kindlin-3 is essential for integrin activation and platelet aggregation Markus Moser, Bernhard Nieswandt, Siegfried Ussar, Nature Medicine ThrombopoiesisThrombosisBlood PlateletHematologyVascular Biology	2008	681
SILAC Mouse for Quantitative Proteomics Uncovers Kindlin-3 as an Essential Factor for Red Blood Cell Function Marcus Krüger, Markus Moser, Siegfried Ussar, Cell Knockout MouseEssential FactorAutoimmune DiseaseMedicineHematology	2008	667
Altered circadian activity rhythms and sleep in mice devoid of prion protein Irene Tobler, Stephanie E. Gaus, Tom Deboer, Nature SleepMolecular NeuroscienceMice DevoidMedicinePhysiology	1996	653
Prion (PrPSc)-specific epitope defined by a monoclonal antibody Carsten Korth, B. Stierli, P. Streit, Nature ImmunologyPathologyPrion DiseaseAntigen ProcessingMedicine	1997	563
Kindlin-2 controls bidirectional signaling of integrins Eloi Montañez, Siegfried Ussar, Martina Schifferer, Genes & Development Cell AdhesionCytoskeletonCellular PhysiologyEndocytic PathwayMatrix Biology	2008	436
Leukocyte adhesion deficiency-III is caused by mutations in KINDLIN3 affecting integrin activation Lena Svensson, Kimberley Howarth, Alison McDowall, Nature Medicine Autoimmune DiseaseCell AdhesionLeukocyte Adhesion Deficiency-iiiHematologyImmunology	2009	408
Kindlin-3 is required for β2 integrin–mediated leukocyte adhesion to endothelial cells Markus Moser, Martina Bauer, Stephan Schmid, Nature Medicine Endothelial CellsCell SignalingCell AdhesionImmunologyBlood Cell	2009	358
Developmental expression of the prion protein gene in glial cells Markus Moser, Raymond J. Colello, Uwe Pott, Neuron Developmental BiologyGeneticsPrion DiseaseMolecular GeneticsMolecular Neurobiology	1995	321
Novel function for blood platelets and podoplanin in developmental separation of blood and lymphatic circulation Pavel Uhrín, Jan Zaujec, Johannes M. Breuss, Blood	2010	291

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