Stephanie Markovina - Concepedia

Concepedia

Author

Stephanie Markovina

University of Wisconsin–Madison

Also Known As

S Markovina, S. Markovina, Stephanie Markovina, Stephanie S. Markovina, Stephanie Subasic Markovina

52

Publications

1.1K

Citations

14

H-Index

32

Concepts

All Affiliations

University of Wisconsin–Madison University of Wisconsin Carbone Cancer Center National Cancer Institute Center for Cancer Research UW Health University Hospital

SM

About

Stephanie Markovina is an author at University of Wisconsin–Madison specializing in medicine, cell biology, and radiation oncology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	597
2	Cell Biology	5	5	578
3	Natural Sciences	2	2	215
4	Immunology	2	2	78
5	Health Sciences	1	1	19

Stephanie Markovina

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15

Publications

1.1K

Citations

14

H-Index

	Year	Citations
Thioredoxin nuclear translocation and interaction with redox factor-1 activates the activator protein-1 transcription factor in response to ionizing radiation. Wei Sj, A.C. Botero, Kiichi Hirota, PubMed Thioredoxin Nuclear TranslocationRadiation EffectRadiation ExposureMolecular BiologyRadiation Biology	2000	204
Geldanamycin and 17-allylamino-17-demethoxygeldanamycin potentiate the in vitro and in vivo radiation response of cervical tumor cells via the heat shock protein 90-mediated intracellular signaling and cytotoxicity. Kheem S. Bisht, C. Matthew Bradbury, David Mattson, PubMed Heat Shock ProteinTumor Biology17-Allylamino-17-demethoxygeldanamycin PotentiateOncologyCervical Cancer	2003	178
Bortezomib-Resistant Nuclear Factor-κB Activity in Multiple Myeloma Cells Stephanie Markovina, Natalie S. Callander, Shelby L. O’Connor, Molecular Cancer Research	2008	144
Thioredoxin reductase regulates AP-1 activity as well as thioredoxin nuclear localization via active cysteines in response to ionizing radiation Shervin Karimpour, Junyang Lou, Lilie L. Lin, Oncogene Reductive StressSignal TransductionRedox RegulatorRadiation BiologyAp-1 Activity	2002	118
Bone marrow stromal cells from multiple myeloma patients uniquely induce bortezomib resistant NF-κB activity in myeloma cells Stephanie Markovina, Natalie S. Callander, Shelby L. O’Connor, Molecular Cancer	2010	117
Indomethacin-induced radiosensitization and inhibition of ionizing radiation-induced NF-kappaB activation in HeLa cells occur via a mechanism involving p38 MAP kinase. C. Matthew Bradbury, Stephanie Markovina, Simin Wei, PubMed P38 Map KinaseRadiation EffectImmunologyRadiation ExposureCell Death	2001	67
Prevalence of bortezomib-resistant constitutive NF-kappaB activity in mantle cell lymphoma David T. Yang, Ken H. Young, Brad S. Kahl, Molecular Cancer	2008	61
DNMT1 as a Molecular Target in a Multimodality-Resistant Phenotype in Tumor Cells Mark V. Mishra, Kheem S. Bisht, Lunching Sun, Molecular Cancer Research	2008	42
Hyaluronan and proteoglycan link protein 1 (HAPLN1) activates bortezomib-resistant NF-κB activity and increases drug resistance in multiple myeloma Mailee Huynh, Chorom Pak, Stephanie Markovina, Journal of Biological Chemistry	2017	42
Acetyl-l-carnitine (ALCAR) for the prevention of chemotherapy-induced peripheral neuropathy in patients with relapsed or refractory multiple myeloma treated with bortezomib, doxorubicin and low-dose dexamethasone: a study from the Wisconsin Oncology Network Natalie S. Callander, Stephanie Markovina, Jens C. Eickhoff, Cancer Chemotherapy and Pharmacology	2014	35

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