Jonathan D. Licht - Concepedia

Author

Jonathan D. Licht

Also Known As

J D Licht, J Licht, J. D Licht, J. D. Licht, J. Licht, J.D. Licht, JD Licht, Jonathan D Licht, Jonathan D. Licht, Jonathan Licht

579

Publications

31K

Citations

H-Index

154

Concepts

All Affiliations

Northwestern University Icahn School of Medicine at Mount Sinai University of Florida Health UF Health Cancer Center Dana-Farber Cancer Institute

About

Jonathan D. Licht is an author at Northwestern University specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	49	89	16.1K
2	Natural Sciences	19	21	1.6K
3	Health Sciences	4	4	92
4	Public Health	3	3	47
5	Engineering	1	1	13

Jonathan D. Licht

197

Publications

27.3K

Citations

H-Index

Page 1

	Year	Citations
Leukemic IDH1 and IDH2 Mutations Result in a Hypermethylation Phenotype, Disrupt TET2 Function, and Impair Hematopoietic Differentiation María E. Figueroa, Omar Abdel‐Wahab, Chao Lü, Cancer Cell Hematological MalignancySomatic VariantHypermethylation PhenotypeMedicineGenetics	2010	2.7K
MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts Thomas Thum, Carina Gross, Jan Fiedler, Nature FibrosisMicrorna-21 ContributesMyocardial DiseasePathologyVascular Biology	2008	2.3K
Mitochondria Are Required for Antigen-Specific T Cell Activation through Reactive Oxygen Species Signaling Laura A. Sena, Sha Li, Amit Jairaman, Immunity InflammationAutoimmune DiseaseMitochondrial FunctionT-regulatory CellImmunology	2013	1.3K
Somatic mutations in PTPN11 in juvenile myelomonocytic leukemia, myelodysplastic syndromes and acute myeloid leukemia Marco Tartaglia, Charlotte M. Niemeyer, Alessandra Fragale, Nature Genetics Hematological MalignancyChromatinSomatic VariantSomatic MutationsMedicine	2003	1K
Deconstructing a Disease: RAR, Its Fusion Partners, and Their Roles in the Pathogenesis of Acute Promyelocytic Leukemia Ari Melnick, Jonathan D. Licht Blood	1999	1K
EZH2 Is Required for Germinal Center Formation and Somatic EZH2 Mutations Promote Lymphoid Transformation Wendy Béguelin, Relja Popovic, Matt Teater, Cancer Cell ChromatinDevelopmental BiologyGerm CellSomatic VariantGerminal Center Formation	2013	828
Transcription Factors, Normal Myeloid Development, and Leukemia Daniel G. Tenen, Robert Hromas, Jonathan D. Licht, Blood	1997	713
Transcription Factors, Normal Myeloid Development, and Leukemia Daniel G. Tenen, Robert Hromas, Jonathan D. Licht, Blood ChromatinDevelopmental BiologyMixed-phenotype Acute LeukemiaTranscription FactorsMedicine	1997	645
Arrest of the cell cycle by the tumour-suppressor BRCA1 requires the CDK-inhibitor p21WAF1/CiPl Kumaravel Somasundaram, Hongbing Zhang, Yi‐Xin Zeng, Nature Cancer ResearchOncologyCell RegulationMedicineCancer Cell Biology	1997	507
Sprouty1 Is a Critical Regulator of GDNF/RET-Mediated Kidney Induction M. Albert Basson, Simge Akbulut, Judy Watson-Johnson, Developmental Cell UrologyRenal FunctionMedicineGdnf/ret-mediated Kidney InductionRenal Inflammation	2005	357

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