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Modeling Smith-Lemli-Opitz syndrome with induced pluripotent stem cells reveals a causal role for Wnt/β-catenin defects in neuronal cholesterol synthesis phenotypes

Smith-lemli-opitz SyndromeInduced Pluripotent Stem CellDevelopmental BiologyOxysterolMedicineWnt/β-catenin DefectsStem Cell ResearchCausal RoleOrganogenesisStem Cell BiologyStem CellsCell BiologyNeural Stem CellCell DevelopmentHealth Sciences