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Antioxidant effects of diallyl trisulfide on high glucose-induced apoptosis are mediated by the PI3K/Akt-dependent activation of Nrf2 in cardiomyocytes

Cardiovascular ToxicityBiochemistryAntioxidant EffectsDiallyl TrisulfideDiabetesApoptosisPhysiologyCell DeathMedicineLipid PeroxidationRedox BiologyHigh Glucose-induced ApoptosisReactive Oxygen SpecieMetabolismPharmacologyCell SignalingOxidative Stress