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Analysis of the individual and aggregate genetic contributions of previously identified serine peptidase inhibitor Kazal type 5 (SPINK5), kallikrein-related peptidase 7 (KLK7), and filaggrin (FLG) polymorphisms to eczema risk

MedicineGeneticsGenetic EpidemiologyHuman PolymorphismGenetic FactorPharmacogenomicsAggregate Genetic ContributionsPharmacologyImmune-related Gene PolymorphismGenetic MedicineClinical GeneticsKallikrein-related Peptidase 7