Publication | Open Access

Activation of PPARβ/δ protects cardiac myocytes from oxidative stress-induced apoptosis by suppressing generation of reactive oxygen/nitrogen species and expression of matrix metalloproteinases

Cardiac MuscleApoptosisCell DeathOxidative Stress-induced ApoptosisCell Death MechanismsRedox BiologyOxidative StressCell SignalingCardiologyMolecular SignalingRedox SignalingMolecular PhysiologyBiochemistryCardiac MyocytesReactive Oxygen SpecieCell BiologyReactive Oxygen/nitrogen SpeciesCardiovascular DiseasePhysiologyMedicine