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Human O-sulfated metabolites of (−)-epicatechin and methyl-(−)-epicatechin are poor substrates for commercial aryl-sulfatases: Implications for studies concerned with quantifying epicatechin bioavailability

BiosynthesisBiochemistryMedicineBioanalysisPoor SubstratesMetabolomic ProfilingEpicatechin BioavailabilityMetabolic ProfilingMetabolomicsMetabolismPharmacologyEnzymatic ModificationHuman O-sulfated Metabolites