Publication | Open Access
A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy
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Citations
39
References
2002
Year
Cancer TherapyLifespan ExtensionAgingMedicineP16ink4a ContributesCellular SenescenceSenescence ProgramTumor SuppressorCancer TreatmentRadiation OncologyOncologyCell BiologyCancer ResearchTumor Biology
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