Helicobacter pylori VacA-induced Inhibition of GSK3 through the PI3K/Akt Signaling Pathway

References

Page 1

	Year	Citations
NF-κB is a target of AKT in anti-apoptotic PDGF signalling Julia A. Romashkova, Sergei S. Makarov Nature Signaling PathwayReceptor Tyrosine KinaseImmunologyCell DeathPathology	1999	1.8K
Requirement for glycogen synthase kinase-3β in cell survival and NF-κB activation Klaus P. Hoeflich, Juan Luo, Elizabeth A. Rubie, Nature Signal TransductionSignaling PathwayMolecular PathwayCell RegulationCell Death	2000	1.4K
Gene structure of the Helicobacter pylori cytotoxin and evidence of its key role in gastric disease. John L. Telford, P Ghiara, M. Dell’Orco, The Journal of Experimental Medicine Microbial ToxinDysbiosisHelicobacter Pylori CytotoxinGastric DiseaseMedicine	1994	591
β-catenin–sensitive isoforms of lymphoid enhancer factor-1 are selectively expressed in colon cancer Karine Hovanes, Tony W.H. Li, Jesus E. Munguia, Nature Genetics Tumor BiologyGastrointestinal OncologyMedicineColorectal CancerCancer Cell Biology	2001	463
Helicobacter pylori CagA interacts with E-cadherin and deregulates the β-catenin signal that promotes intestinal transdifferentiation in gastric epithelial cells Yo Kurashima, Yasuhiro Teishikata, Yukie Yamahashi, Oncogene Signal TransductionGastrointestinal Pharmacologyβ-Catenin SignalGastroenterologyPathology	2007	455
beta-Catenin mutation is a frequent cause of Wnt pathway activation in gastric cancer. Wilson M. Clements, Jiang Wang, Amod A. Sarnaik, PubMed Tumor BiologyGastrointestinal OncologyMedicineGastroenterologyCancer Cell Biology	2002	364
Control of β-Catenin Stability Adrian Salic, Ethan Lee, Leslie Mayer, Molecular Cell β-Catenin StabilitySystem StabilityBiophysicsStability AnalysisStability	2000	341
Inhibition of Wnt signaling by ICAT, a novel β-catenin-interacting protein K. Tago, Tsutomu Nakamura, Michiru Nishita, Genes & Development	2000	335
The <i>Helicobacter pylori</i> Vacuolating Toxin Inhibits T Cell Activation by Two Independent Mechanisms Marianna Boncristiano, Silvia Rossi Paccani, Silvia Barone, The Journal of Experimental Medicine InflammationMicrobial ToxinSignal TransductionIndependent MechanismsSignaling Pathway	2003	302
Formation of anion-selective channels in the cell plasma membrane by the toxin VacA of Helicobacter pylori is required for its biological activity Ildikò Szabó The EMBO Journal Signal TransductionBiochemistryMedicineMembrane TransportCell Plasma Membrane	1999	272

Page 1