Jonathan A. Deane - Concepedia

Author

Jonathan A. Deane

Genomics Institute of the Novartis Research Foundation

Also Known As

Deane, Jonathan, Jonathan A Deane, Jonathan A. Deane, Jonathan Deane

Publications

2.6K

Citations

H-Index

Concepts

All Affiliations

Genomics Institute of the Novartis Research Foundation University of California, Irvine Novartis (Switzerland)Beth Israel Deaconess Medical Center National Institute of Allergy and Infectious Diseases

About

Jonathan A. Deane is an author at Genomics Institute of the Novartis Research Foundation specializing in medicine, immunology, and autoimmunity.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	900
2	Allergy	2	2	148
3	Cell Biology	2	2	740
4	Natural Sciences	1	1	126
5	Cellular Physiology	1	1	126

Jonathan A. Deane

Publications

2.2K

Citations

H-Index

Page 1

	Year	Citations
Autoreactive B Cell Responses to RNA-Related Antigens Due to <i>TLR7</i> Gene Duplication Prapaporn Pisitkun, Jonathan A. Deane, Michael J. Difilippantonio, Science	2006	884
Control of Toll-like Receptor 7 Expression Is Essential to Restrict Autoimmunity and Dendritic Cell Proliferation Jonathan A. Deane, Prapaporn Pisitkun, Rebecca S. Barrett, Immunity InflammationToll-like ReceptorsToll-like Receptor 7Autoimmune DiseaseDendritic Cell Proliferation	2007	614
Role of Phosphoinositide 3-Kinase Regulatory Isoforms in Development and Actin Rearrangement Saskia M. Brachmann, Claudine M. Yballe, Metello Innocenti, Molecular and Cellular Biology Molecular RegulationGeneticsCytoskeletonP85 Regulatory SubunitsCellular Physiology	2005	126
Dual signaling by innate and adaptive immune receptors is required for TLR7-induced B-cell–mediated autoimmunity Elizabeth R. Walsh, Prapaporn Pisitkun, Elisaveta Voynova, Proceedings of the National Academy of Sciences	2012	90
Phosphoinositide 3-kinase signaling is essential for ABL oncogene–mediated transformation of B-lineage cells Michael G. Kharas, Jonathan A. Deane, Stephane Wong, Blood	2004	85
<i>Ifih1</i> Gene Dose Effect Reveals MDA5-Mediated Chronic Type I IFN Gene Signature, Viral Resistance, and Accelerated Autoimmunity Steve P. Crampton, Jonathan A. Deane, Lionel Feigenbaum, The Journal of Immunology ImmunodeficienciesImmune RegulationImmunologyImmunodominanceImmunologic Mechanism	2011	84
Enhanced T Cell Proliferation in Mice Lacking the p85β Subunit of Phosphoinositide 3-Kinase Jonathan A. Deane, Matthew J. Trifilo, Claudine M. Yballe, The Journal of Immunology	2004	69
Sjögren's syndrome-like disease in mice with T cells lacking class 1A phosphoinositide-3-kinase Jean Oak, Jonathan A. Deane, Michael G. Kharas, Proceedings of the National Academy of Sciences T-regulatory CellImmunologyImmune RegulationPathologyImmunologic Mechanism	2006	64
T-cell function is partially maintained in the absence of class IA phosphoinositide 3-kinase signaling Jonathan A. Deane, Michael G. Kharas, Jean Oak, Blood	2006	54
Class IA Phosphoinositide 3-Kinase Modulates Basal Lymphocyte Motility in the Lymph Node Melanie P. Matheu, Jonathan A. Deane, Ian Parker, The Journal of Immunology	2007	38

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