Concepedia

Author

Eva Petermann

University of BirminghamORCID

Also Known As

E Petermann, E. Petermann, Eva Petermann, James F. Beesley, Petermann, Eva

91

Publications

8.1K

Citations

34

H-Index

40

Concepts

All Affiliations

University of BirminghamUniversity of OxfordUniversity of SussexCRUK/MRC Oxford Institute for Radiation OncologyStockholm University
EP

About

Eva Petermann is an author at University of Birmingham specializing in medicine, natural sciences, and molecular biology.

Top Concepts

#ConceptH-IndexPublicationsCitations

1

Medicine

12

12

1.5K

2

Natural Sciences

10

10

1.3K

3

Cell Biology

8

8

1.2K

4

Biochemistry

2

2

45

5

Health Sciences

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Eva Petermann

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32

Publications

4.8K

Citations

27

H-Index

YearCitations

Hydroxyurea-Stalled Replication Forks Become Progressively Inactivated and Require Two Different RAD51-Mediated Pathways for Restart and Repair

Eva Petermann, Manuel Luís Orta, Natalia Issaeva,

Molecular Cell

2010

824

PARP is activated at stalled forks to mediate Mre11‐dependent replication restart and recombination

Helen E. Bryant, Eva Petermann, Niklas Schultz,

The EMBO Journal

2009

589

ATR‐dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling

Thomas Stiff, Sarah Walker, Karen Cerosaletti,

The EMBO Journal

2006

351

Increased global transcription activity as a mechanism of replication stress in cancer

Panagiotis Kotsantis, Lara Marques Silva, Sarah Irmscher,

Nature Communications

2016

339

Chk1 promotes replication fork progression by controlling replication initiation

Eva Petermann, Mick Woodcock, Thomas Helleday

Proceedings of the National Academy of Sciences

2010

292

Increased replication initiation and conflicts with transcription underlie Cyclin E-induced replication stress

Rebecca M. Jones, Oliver Mortusewicz, Irfan Afzal,

Oncogene

2012

284

ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells

Marwan Kwok, Nicholas Davies, Angelo Agathanggelou,

Blood

2015

266

Chk1 regulates the density of active replication origins during the vertebrate S phase

Apolinar Maya‐Mendoza, Eva Petermann, David A. Gillespie,

The EMBO Journal

2007

230

PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation

George E. Ronson, Ann Liza Piberger, Martin R. Higgs,

Nature Communications

2018

209

Chk1 Requirement for High Global Rates of Replication Fork Progression during Normal Vertebrate S Phase

Eva Petermann, Apolinar Maya‐Mendoza, George Zachos,

Molecular and Cellular Biology

2006

175

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