Chun‐Ling Dai - Concepedia

Author

Chun‐Ling Dai

New York State Office for People With Developmental Disabilities ORCID

Also Known As

Chun-Ling Dai, Chun-ling Dai, Chun-ling Dai (2164816), ChunLing Dai, Chunling Dai, Chun‐Ling Dai, Chun‐ling Dai, Dai Chun-ling

Publications

2.3K

Citations

H-Index

Concepts

All Affiliations

New York State Office for People With Developmental Disabilities Nantong University Union Hospital Hubei University of Science and Technology Second Affiliated Hospital of Zhejiang University

About

Chun‐Ling Dai is an author at New York State Office for People With Developmental Disabilities specializing in medicine, neuroscience, and alzheimer's disease.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	14	1.1K
2	Social Sciences	6	6	610
3	Health Sciences	2	2	127
4	Biochemistry	2	2	119
5	Anesthesia	1	1	34

Chun‐Ling Dai

Publications

1.9K

Citations

H-Index

Page 1

	Year	Citations
A Non-transgenic Mouse Model (icv-STZ Mouse) of Alzheimer’s Disease: Similarities to and Differences from the Transgenic Model (3xTg-AD Mouse) Yanxing Chen, Zhihou Liang, Julie Blanchard, Molecular Neurobiology Neurodegenerative DiseasesAlzheimer's DiseaseNeurobiology Of DiseaseTransgenic Mouse ModelsRodent Models	2012	284
Tau passive immunization inhibits not only tau but also Aβ pathology Chun‐Ling Dai, Yunn Chyn Tung, Fei Liu, Alzheimer s Research & Therapy	2017	187
Intranasal insulin restores insulin signaling, increases synaptic proteins, and reduces Aβ level and microglia activation in the brains of 3xTg-AD mice Yanxing Chen, Zhao Yang, Chun‐Ling Dai, Experimental Neurology Neurodegenerative DiseasesNeuroregenerationSynaptic PlasticityInsulin SignalingSynaptic Signaling	2014	118
Intracerebroventricular Streptozotocin Exacerbates Alzheimer-Like Changes of 3xTg-AD Mice Yanxing Chen, Zhihou Liang, Zhu Tian, Molecular Neurobiology Alzheimer's DiseaseMedicineNeuropharmacologyNeuroprotectionNeuroscience	2013	106
Developmental Regulation of Protein O-GlcNAcylation, O-GlcNAc Transferase, and O-GlcNAcase in Mammalian Brain Ying Liu, Xiaojing Li, Yang Yu, PLoS ONE	2012	102
Chronic cerebral hypoperfusion causes decrease of O-GlcNAcylation, hyperphosphorylation of tau and behavioral deficits in mice Zhao Yang, Jin‐Hua Gu, Chun‐Ling Dai, Frontiers in Aging Neuroscience AgingAlzheimer's DiseaseMemoryBrain InjuryNeurology	2014	96
Tau passive immunization blocks seeding and spread of Alzheimer hyperphosphorylated Tau-induced pathology in 3 × Tg-AD mice Chun‐Ling Dai, Wen Hu, Yunn Chyn Tung, Alzheimer s Research & Therapy	2018	91
O‐Glc<scp>NA</scp>cylation of protein kinase A catalytic subunits enhances its activity: a mechanism linked to learning and memory deficits in Alzheimer's disease Shu-Tao Xie, Nana Jin, Jianlan Gu, Aging Cell Synaptic TransmissionGlycobiologySynaptic SignalingSocial SciencesMemory Deficits	2016	88
Passive immunization targeting the N-terminal projection domain of tau decreases tau pathology and improves cognition in a transgenic mouse model of Alzheimer disease and tauopathies Chun‐Ling Dai, Xia Chen, Syed Faraz Kazim, Journal of Neural Transmission Knockout MouseAlzheimer's DiseaseTransgenic Mouse ModelsMedicinePassive Immunization	2014	87
Disease modifying effect of chronic oral treatment with a neurotrophic peptidergic compound in a triple transgenic mouse model of Alzheimer's disease Syed Faraz Kazim, Julie Blanchard, Chun‐Ling Dai, Neurobiology of Disease Alzheimer's DiseaseMedicineDementiaChronic Oral TreatmentNeuropharmacology	2014	81

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