Roger R. Reddel - Concepedia

Author

Roger R. Reddel

Children's Medical Research Institute ORCID

Also Known As

Chris Worthley, Christopher L. Wolfgang, Hilda A. Pickett, Qing Zhong, R R Reddel, R Reddel, R. R. Reddel, R. Reddel, R. Reddel, Roger, R.R. Reddel

540

Publications

29.2K

Citations

H-Index

163

Concepts

All Affiliations

Children's Medical Research Institute The University of Sydney National Institute of Advanced Industrial Science and Technology Children's Hospital at Westmead National Cancer Institute

About

Roger R. Reddel is an author at Children's Medical Research Institute specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	65	108	14.6K
2	Natural Sciences	20	22	2.8K
3	Health Sciences	6	6	681
4	Engineering	4	4	159
5	Arts	1	1	188

Roger R. Reddel

180

Publications

21.8K

Citations

H-Index

Page 1

	Year	Citations
Evidence for an alternative mechanism for maintaining telomere length in human tumors and tumor-derived cell lines Tracy M. Bryan, Anna Englezou, Luciano Dalla‐Pozza, Nature Medicine Tumor BiologyAlternative MechanismMedicineTelomere LengthPathology	1997	1.3K
Telomere elongation in immortal human cells without detectable telomerase activity. Tracy M. Bryan, Anna Englezou, Jeena Gupta, The EMBO Journal Genome InstabilityMolecular BiologyCellular SenescenceTelomere ElongationMedicine	1995	1.3K
Telomere maintenance by recombination in human cells Melissa A. Dunham, Axel A. Neumann, Clare L. Fasching, Nature Genetics Genome InstabilityHuman CellsDna ReplicationChromosomal RearrangementRecombination Dynamic	2000	860
Transformation of human bronchial epithelial cells by infection with SV40 or adenovirus-12 SV40 hybrid virus, or transfection via strontium phosphate coprecipitation with a plasmid containing SV40 early region genes. Roger R. Reddel, B I Gerwin, M G McMenamin, PubMed Viral ReplicationCancer-associated VirusSv40-transformed CellsPathogenesisViral Pathogenesis	1988	788
Telomerase-negative immortalized human cells contain a novel type of promyelocytic leukemia (PML) body. Thomas R. Yeager, Axel A. Neumann, Anna Englezou, PubMed Hematological MalignancyChromatinGenome InstabilityPromyelocytic LeukemiaNovel Type	1999	691
Protein Composition of Catalytically Active Human Telomerase from Immortal Cells Scott B. Cohen, Mark E. Graham, George O. Lovrecz, Science	2007	657
Loss of ATRX, Genome Instability, and an Altered DNA Damage Response Are Hallmarks of the Alternative Lengthening of Telomeres Pathway Courtney A. Lovejoy, Wen‐Di Li, Steven Reisenweber, PLoS Genetics	2012	618
Isolation of a Candidate Human Telomerase Catalytic Subunit Gene, Which Reveals Complex Splicing Patterns in Different Cell Types Andrzej Kilian, David D.L. Bowtell, Helen E. Abud, Human Molecular Genetics	1997	587
DNA C-circles are specific and quantifiable markers of alternative-lengthening-of-telomeres activity Jeremy D. Henson, Ying Cao, Lily I. Huschtscha, Nature Biotechnology ChromatinGenome InstabilityDnaCell DivisionDna C-circles	2009	488
Alternative Lengthening of Telomeres Is Characterized by High Rates of Telomeric Exchange Arturo Londoño‐Vallejo, Héra Der‐Sarkissian, Lucien Cazes, Cancer Research	2004	310

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