Jamal Nasir - Concepedia

Author

Jamal Nasir

Also Known As

Alessandra Renieri, Chloe Kirk, Ioannis Karakikes, J Nasir, J. Nasir, Jamal Abdul Nasir, Jamal Abdul Nasir, Jamal Nasir, Joohi Nasir, Nasir, J

144

Publications

Citations

H-Index

122

Concepts

All Affiliations

University of British Columbia St George's, University of London Hospital for Sick Children University of Edinburgh King Abdulaziz University

About

Jamal Nasir is an author at University of British Columbia specializing in medicine, genetics, and molecular genetics.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Engineering	2	2	24
2	Arts	1	1	53
3	Education	1	1	42
4	Health Sciences	1	1	33
5	Language Studies	1	1	53

Jamal Nasir

Publications

5.4K

Citations

H-Index

Page 1

	Year	Citations
Targeted disruption of the Huntington's disease gene results in embryonic lethality and behavioral and morphological changes in heterozygotes Jamal Nasir, Stan Floresco, John R. OʼKusky, Cell Knockout MouseMorphological ChangesDevelopmental BiologyEmbryonic LethalityGenetic Disorder	1995	861
A YAC Mouse Model for Huntington’s Disease with Full-Length Mutant Huntingtin, Cytoplasmic Toxicity, and Selective Striatal Neurodegeneration John Hodgson, N. Agopyan, Claire‐Anne Gutekunst, Neuron Neurodegenerative DiseasesFull-length Mutant HuntingtinSelective Striatal NeurodegenerationGeneticsYac Mouse Model	1999	828
A gene encoding a putative GTPase regulator is mutated in familial amyotrophic lateral sclerosis 2 Collette K. Hand, Hitoshi Osuga, Yoshiko Yanagisawa, Nature Genetics Neurodegenerative DiseasesAmyotrophic Lateral SclerosisGeneticsExperimental NeuropathologyPutative Gtpase Regulator	2001	675
Paraptosis: mediation by MAP kinases and inhibition by AIP-1/Alix Sabina Sperandio, Karen S. Poksay, Ian de Belle, Cell Death and Differentiation Signal TransductionSignaling PathwayMap KinasesReceptor Tyrosine KinaseMedicine	2004	379
Cell death attenuation by `Usurpin', a mammalian DED-caspase homologue that precludes caspase-8 recruitment and activation by the CD-95 (Fas, APO-1) receptor complex Dita Rasper, John P. Vaillancourt, Vicky M. Houtzager, Cell Death and Differentiation Cell Death AttenuationSignal TransductionSignaling PathwayCell InteractionApoptosis	1998	315
GA4GH: International policies and standards for data sharing across genomic research and healthcare Heidi L. Rehm, Angela Page, Lindsay Smith, Cell Genomics	2021	273
Interaction between chromatin proteins MECP2 and ATRX is disrupted by mutations that cause inherited mental retardation Xinsheng Nan, Jianghui Hou, Alan Maclean, Proceedings of the National Academy of Sciences Epigenetic ChangeGeneticsMecp2-atrx InteractionChromatin Proteins Mecp2Atrx Localization	2007	254
CMIP and ATP2C2 Modulate Phonological Short-Term Memory in Language Impairment Dianne F. Newbury, Laura Winchester, Laura Addis, The American Journal of Human Genetics	2009	191
Absence of Disease Phenotype and Intergenerational Stability of the Cag Repeat in Transgenic Mice Expressing the Human Huntington Disease Transcript Y. Paul Goldberg, Michael A. Kalchman, Martina Metzler, Human Molecular Genetics	1996	112
Differential 3′ polyadenylation of the Huntington disease gene results in two mRNA species with variable tissue expression B. Lin, Johanna M. Rommens, Rona K. Graham, Human Molecular Genetics GeneticsMolecular BiologyGene CharacterizationMolecular GeneticsDisease Gene Identification	1993	96

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