Publication | Closed Access
GDF15 attenuates sepsis-induced myocardial dysfunction by inhibiting cardiomyocytes ferroptosis via the SOCS1/GPX4 signaling pathway
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Citations
44
References
2024
Year
InflammationCardiomyopathyHeart FailureMolecular PhysiologyFibrosisSignal TransductionImmunologySepsis-induced Myocardial DysfunctionCardiologyCardiomyocytes FerroptosisMedicineCell BiologyCell Signaling
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