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Hyperoside attenuates Cd-induced kidney injury via inhibiting NLRP3 inflammasome activation and ROS/MAPK/NF-κB signaling pathway in vivo and in vitro

InflammationAnti-inflammatoryImmunologyImmune RegulationCell DeathChronic InflammationRenal InflammationNlrp3 Inflammasome ActivationRenal PathophysiologyCd-induced Kidney InjuryAcute Kidney InjuryMedicineCell BiologyCell SignalingNephrologyKidney ResearchKidney Tubule Remodeling