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Long-term exposure to PM2.5 aggravates pulmonary fibrosis and acute lung injury by disrupting Nrf2-mediated antioxidant function

InflammationAcute Lung InjuryInflammatory Lung DiseaseAllergyLung InflammationNrf2-mediated Antioxidant FunctionMedicineRespiratory ToxicologyCell DeathPulmonary FibrosisReactive Oxygen SpeciePharmacologyPulmonary DiseaseLong-term ExposureOxidative StressInhalation Toxicology