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Promoting effect of baicalin on nitric oxide production in CMECs via activating the PI3K-AKT-eNOS pathway attenuates myocardial ischemia–reperfusion injury

InflammationNitric Oxide ProductionMolecular PhysiologyBiochemistryReactive Nitrogen SpecieCell SignalingPi3k-akt-enos PathwayPhysiologyMedicineMyocardial Ischemia–reperfusion InjuryEndothelial DysfunctionVascular BiologyPharmacologyCardiologyNitrosative StressOxidative Stress