Familial knockin mutation of LRRK2 causes lysosomal dysfunction and accumulation of endogenous insoluble α-synuclein in neurons - Concepedia

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Familial knockin mutation of LRRK2 causes lysosomal dysfunction and accumulation of endogenous insoluble α-synuclein in neurons

DOI Full Paper Access

138

Citations

73

References

2017

Year

Jason Schapansky, Saurabh Khasnavis, Mark P. DeAndrade, Jonathan Nardozzi, Samuel R. Falkson, Justin D. Boyd, John Sanderson, Tim Bartels, Heather L. Melrose, Matthew J. LaVoie

Neurobiology of Disease

Neurodegenerative DiseasesMolecular NeuroscienceFamilial Knockin MutationSynaptic SignalingGenetic DisorderMendelian DisorderGeneticsLysosomal DysfunctionDegenerative PathologyMolecular NeurobiologyNeurodegenerationEndogenous Insoluble α-SynucleinMedicineCell BiologyNeurogenetics

References

	Year	Citations

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