Publication | Open Access
A tripartite complex of suPAR, APOL1 risk variants and αvβ3 integrin on podocytes mediates chronic kidney disease
199
Citations
42
References
2017
Year
GlomerulonephritisApol1 Risk VariantsMedicineKidney FailureRenal PathologyImmunologyRenal InflammationTripartite ComplexRenal PathophysiologyChronic Kidney DiseaseCell BiologyCell SignalingNephrologyKidney Research
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