David J. Friedman - Concepedia

Author

David J. Friedman

Beth Israel Deaconess Medical Center ORCID

Also Known As

D Friedman, D J Friedman, D. Friedman, D. J. Friedman, D.J. Friedman, DAVID FRIEDMAN, DAVID J. FRIEDMAN, David J. Friedman, David Friedman, David J Friedman

184

Publications

13.7K

Citations

H-Index

137

Concepts

All Affiliations

Beth Israel Deaconess Medical Center Harvard University Broad Institute Center for Vascular Biology Research Boston University

About

David J. Friedman is an author at Beth Israel Deaconess Medical Center specializing in medicine, chronic kidney disease, and nephrology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	24	32	5.7K
2	Natural Sciences	5	5	171
3	Health Sciences	3	3	104
4	Business	1	1	26
5	Engineering	1	1	26

David J. Friedman

Publications

12.2K

Citations

H-Index

Page 1

	Year	Citations
Adenosine generation catalyzed by CD39 and CD73 expressed on regulatory T cells mediates immune suppression Silvia Deaglio, Karen M. Dwyer, Wenda Gao, The Journal of Experimental Medicine	2007	2.3K
Association of Trypanolytic ApoL1 Variants with Kidney Disease in African Americans Giulio Genovese, David J. Friedman, Michael D. Ross, Science GlomerulonephritisUrologyRenal FunctionKidney ResearchMedicine	2010	2K
A Controlled Trial of Fluconazole to Prevent Fungal Infections in Patients Undergoing Bone Marrow Transplantation Jesse L. Goodman, Drew J. Winston, Ronald A. Greenfield, New England Journal of Medicine	1992	1.2K
APOL1 Genetic Variants in Focal Segmental Glomerulosclerosis and HIV-Associated Nephropathy Jeffrey B. Kopp, George W. Nelson, Karmini Sampath, Journal of the American Society of Nephrology Glomerular DiseaseTrypanolytic VariantsApol1 VariantsImmunologyGenetic Epidemiology	2011	836
Induction of Apoptosis in Uninfected Lymphocytes by HIV-1 Tat Protein Chiang J. Li, David J. Friedman, Chuanlin Wang, Science T-regulatory CellApoptosisImmunologyCell DeathPathology	1995	577
Innate immunity pathways regulate the nephropathy gene Apolipoprotein L1 Brendan Nichols, Prachi Jog, Jessica H. Lee, Kidney International InflammationGlomerulonephritisAutoimmune DiseaseMedicineLupus Nephritis	2014	347
CD39 and control of cellular immune responses Karen M. Dwyer, Silvia Deaglio, Wenda Gao, Purinergic Signalling Adaptive Immune SystemImmunologyImmune RegulationImmunologic MechanismAntigen Processing	2007	287
CD39 deletion exacerbates experimental murine colitis and human polymorphisms increase susceptibility to inflammatory bowel disease David J. Friedman, Beat Künzli, Yousif I. A-Rahim, Proceedings of the National Academy of Sciences Chronic Inflammatory DiseasesImmunologyGenetic EpidemiologyPathologyGastroenterology	2009	280
Population-Based Risk Assessment of APOL1 on Renal Disease David J. Friedman, Julia Kozlitina, Giulio Genovese, Journal of the American Society of Nephrology	2011	227
APOL1 kidney disease risk variants cause cytotoxicity by depleting cellular potassium and inducing stress-activated protein kinases Opeyemi A. Olabisi, Jia-Yue Zhang, Lynn VerPlank, Proceedings of the National Academy of Sciences	2015	226

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