Publication | Open Access

Peripheral administration of the soluble TNF inhibitor XPro1595 modifies brain immune cell profiles, decreases beta-amyloid plaque load, and rescues impaired long-term potentiation in 5xFAD mice

InflammationNeurodegenerative DiseasesPeripheral AdministrationAutoimmune DiseaseMedicineImmunologyChronic InflammationLong-term PotentiationAutoimmunityNeurochemical BiomarkersNeuroprotectionNeurologyNeuroscienceBeta-amyloid Plaque LoadBrain-immune InteractionNeuroimmunologyNeuroinflammation