Publication | Open Access
The type 2 diabetes presumed causal variant within TCF7L2 resides in an element that controls the expression of ACSL5
80
Citations
22
References
2016
Year
Metabolic SyndromeTcf7l2 ResidesAutoimmune DiseaseDisease MechanismDiabetesGenetic EpidemiologyImmunologyPathologyType 2Causal VariantAutoimmunityMedicineCell SignalingImmune Dysregulation
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