Leucine induces myofibrillar protein accretion in cultured skeletal muscle through m<scp>TOR</scp> dependent and ‐independent control of myosin heavy chain m<scp>RNA</scp> levels

Abstract

This study clearly demonstrates myofibrillar and not generic protein accretion in skeletal muscle following leucine supplementation, and suggests this involves pre-translational control of MyHC expression by leucine.