Tumor Necrosis Factor-α Accelerates the Calcification of Human Aortic Valve Interstitial Cells Obtained from Patients with Calcific Aortic Valve Stenosis via the BMP2-Dlx5 Pathway

References

Page 1

	Year	Citations
Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method Kenneth J. Livak, Thomas D. Schmittgen Methods Functional GenomicsEngineeringMedicineGeneticsGene Recognition	2001	176.9K
A prospective survey of patients with valvular heart disease in Europe: The Euro Heart Survey on Valvular Heart Disease Bernard Iung European Heart Journal	2003	3.5K
Bone Formation and Inflammation in Cardiac Valves Emile R. Mohler, Francis H. Gannon, Carol Reynolds, Circulation	2001	1K
A physical and functional map of the human TNF-α/NF-κB signal transduction pathway Tewis Bouwmeester, Angela Bauch, Heinz Ruffner, Nature Cell Biology InflammationTranscriptional RegulationMolecular PhysiologySignal TransductionSignaling Pathway	2004	1K
Human Aortic Valve Calcification Is Associated With an Osteoblast Phenotype Nalini M. Rajamannan, Malayannan Subramaniam, David J. Rickard, Circulation	2003	708
Lipid Oxidation Products Have Opposite Effects on Calcifying Vascular Cell and Bone Cell Differentiation Farhad Parhami, Andrew D. Morrow, Jennifer P. Balucan, Arteriosclerosis Thrombosis and Vascular Biology	1997	615
Tumor Necrosis Factor-α Promotes In Vitro Calcification of Vascular Cells via the cAMP Pathway Yin Tintut, Jignesh Patel, Farhad Parhami, Circulation	2000	516
Specification of Jaw Subdivisions by <i>Dlx</i> Genes Michael J. Depew, Thomas Lufkin, John L.R. Rubenstein Science	2002	459
Rapidly Forming Apatitic Mineral in an Osteoblastic Cell Line (UMR 106—01 BSP) Clark Stanford, Paul A. Jacobson, E. D. Eanes, Journal of Biological Chemistry	1995	437
Progression of aortic valve stenosis: TGF-β1 is present in calcified aortic valve cusps and promotes aortic valve interstitial cell calcification via apoptosis Jian Bo, Navneet Narula, Quanyi Li, The Annals of Thoracic Surgery MedicinePathologyValve DiseaseVascular BiologyValvular Heart Disease	2003	432

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