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Apigenin attenuates copper-mediated β-amyloid neurotoxicity through antioxidation, mitochondrion protection and MAPK signal inactivation in an AD cell model

Neurodegenerative DiseasesAlzheimer's DiseaseBiochemistryMitochondrion ProtectionMedicineCell DeathAd Cell ModelDegenerative PathologyNeuroprotectionPharmacologyCell BiologyRedox BiologyMapk Signal InactivationOxidative Stress