Publication | Open Access
Double deficiency in IL-17 and IFN-γ signalling significantly suppresses the development of diabetes in the NOD mouse
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Citations
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References
2013
Year
ImmunologyImmune RegulationDouble DeficiencyImmune SystemInsulin SignalingImmune DysregulationInflammationMetabolic SignalingCell SignalingAutoimmune DiseaseAutoimmunityDiabetes ComplicationsCell BiologyInborn Error Of ImmunityNod MouseCytokineDevelopmental BiologyImmune Cell DevelopmentDiabetesMedicineCell Development
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