Antimetastatic potential of fisetin involves inactivation of the PI3K/Akt and JNK signaling pathways with downregulation of MMP-2/9 expressions in prostate cancer PC-3 cells - Concepedia

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Antimetastatic potential of fisetin involves inactivation of the PI3K/Akt and JNK signaling pathways with downregulation of MMP-2/9 expressions in prostate cancer PC-3 cells

144

Citations

32

References

2009

Year

Chi‐Sheng Chien, Kun‐Hung Shen, Jau‐Shyang Huang, Shian-Chin Ko, Yuan‐Wei Shih

Molecular and Cellular Biochemistry

Perineural InvasionFisetin Involves InactivationMedicineMmp-2/9 ExpressionsAntimetastatic PotentialTumor SuppressorProstatic DiseasePharmacologyCell BiologyCell SignalingTumor Microenvironment

References

	Year	Citations

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