Interleukin-1-induced acute bone resorption facilitates the secretion of fibroblast growth factor 23 into the circulation

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FGF-23 Is a Potent Regulator of Vitamin D Metabolism and Phosphate Homeostasis

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Klotho converts canonical FGF receptor into a specific receptor for FGF23

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Evidence for osteocyte regulation of bone homeostasis through RANKL expression

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Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

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