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The SPRY domain of Pyrin, mutated in familial Mediterranean fever patients, interacts with inflammasome components and inhibits proIL-1β processing

InflammationSpry DomainDisease MechanismInhibits Proil-1β ProcessingPathogenesisImmunologyPathologyInflammasomeImmune FunctionInflammasome ComponentsAutoinflammatory DiseaseMedicineCell Signaling