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Increased activation-induced cell death of high IFN-γ–producing TH1 cells as a mechanism of TH2 predominance in atopic diseases

InflammationTh2 PredominanceAtopic DiseasesImmune Cell DevelopmentImmune RegulationImmunologyCell DeathHypersensitivityImmunologic MechanismImmune FunctionActivation-induced Cell DeathCellular Immune ResponseImmunopathologyImmune SystemMedicineCell BiologyCell SignalingImmune Dysregulation