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High glucose-induced apoptosis in human vascular endothelial cells is mediated through NF-κB and c-Jun NH2-terminal kinase pathway and prevented by PI3K/Akt/eNOS pathway

InflammationMedicineApoptosisPhysiologyImmunologyCell DeathEndothelial DysfunctionVascular BiologyDiabetes ComplicationsHigh Glucose-induced ApoptosisPi3k/akt/enos PathwayCell Death MechanismsCell BiologyCell Signaling