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Trovafloxacin-induced replication stress sensitizes HepG2 cells to tumor necrosis factor-alpha-induced cytotoxicity mediated by extracellular signal-regulated kinase and ataxia telangiectasia and Rad3-related

InflammationSignaling PathwayAutoimmune DiseaseHepg2 CellsApoptosisImmunologyAutophagyPathologyCell DeathTrovafloxacin-induced Replication StressAtaxia TelangiectasiaHepatotoxicityMedicineCell BiologyCell SignalingOxidative Stress