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Susceptibility to pulmonary disease due to Mycobacterium avium–intracellulare complex may reflect low IL-17 and high IL-10 responses rather than Th1 deficiency

Inflammatory Lung DiseaseLung InflammationImmunodeficienciesLow Il-17ImmunologyMycobacterium Avium–intracellulare ComplexInfectious Respiratory DiseaseTh1 DeficiencyImmunopathologyImmune SystemMedicineClinical MicrobiologyImmune Dysregulation