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Sensitization of ABCG2-overexpressing cells to conventional chemotherapeutic agent by sunitinib was associated with inhibiting the function of ABCG2

MedicinePhotocarcinogenesisMechanism Of ActionCell DeathAbcg2-overexpressing CellsAnti-cancer AgentConventional Chemotherapeutic AgentPharmacologyCell BiologyCell SignalingTumor MicroenvironmentTumor Biology