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Intrathecal administration of a gap junction decoupler, an inhibitor of Na+–K+–2Cl− cotransporter 1, or a GABAA receptor agonist attenuates mechanical pain hypersensitivity induced by REM sleep deprivation in the rat

Pain DisordersPain MedicineSynaptic TransmissionNeuromodulation TherapiesMolecular PainRem Sleep DeprivationSynaptic SignalingIntrathecal AdministrationPain ManagementHealth SciencesIon ChannelsNeuropharmacologyPharmacologyGap Junction DecouplerPain ResearchNeurophysiologyPhysiologyNeurosciencePain MechanismMedicine