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Mice deficient in BACE1, the Alzheimer's β-secretase, have normal phenotype and abolished β-amyloid generation

Neurodegenerative DiseasesMolecular NeuroscienceAlzheimer's Diseaseβ-Amyloid GenerationMice DeficientDegenerative PathologyProtein MisfoldingNeurologyNeuroscienceNeurodegenerationNormal PhenotypeBrain PathologyMedicineSynaptic SignalingSocial SciencesProtective Mechanisms