Jun‐ichi Hanai - Concepedia

Author

Jun‐ichi Hanai

Beth Israel Deaconess Medical Center

Also Known As

JUN‐ICHI HANAI, Jun-Ichi Hanai, Jun-ichi Hanai, Junichi Hanai, Jun‐Ichi Hanai, Jun‐ichi Hanai

Publications

8.4K

Citations

H-Index

Concepts

All Affiliations

Beth Israel Deaconess Medical Center Harvard University Japan Society for the Promotion of Science Massachusetts Eye and Ear Infirmary Massachusetts General Hospital

About

Jun‐ichi Hanai is an author at Beth Israel Deaconess Medical Center specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	16	6.3K
2	Cell Biology	10	10	4.1K
3	Natural Sciences	4	4	235
4	Cellular Physiology	4	4	626
5	Biochemistry	3	3	221

Jun‐ichi Hanai

Publications

8.2K

Citations

H-Index

Page 1

	Year	Citations
Soluble endoglin contributes to the pathogenesis of preeclampsia Shivalingappa Venkatesha, Mourad Toporsian, Chun Sing Lam, Nature Medicine Soluble Endoglin ContributesGynecologyMaternal HealthMaternal-fetal MedicineEclampsia	2006	1.8K
BMP-7 counteracts TGF-β1–induced epithelial-to-mesenchymal transition and reverses chronic renal injury Michael Zeisberg, Jun‐ichi Hanai, Hikaru Sugimoto, Nature Medicine InflammationGlomerulonephritisUrologyRenal FunctionMedicine	2003	1.3K
Smad6 inhibits signalling by the TGF-β superfamily Takeshi Imamura, Masao Takase, Ayako Nishihara, Nature Molecular PhysiologySignal TransductionSignaling PathwaySmad6 InhibitsMedicine	1997	1K
Targeting Lactate Dehydrogenase-A Inhibits Tumorigenesis and Tumor Progression in Mouse Models of Lung Cancer and Impacts Tumor-Initiating Cells Han Xie, Jun‐ichi Hanai, Jian-Guo Ren, Cell Metabolism OncologyOncogenic AgentImpacts Tumor-initiating CellsMedicineMouse Models	2014	507
Parathyroid Hormone Directs Bone Marrow Mesenchymal Cell Fate Yi Fan, Jun‐ichi Hanai, Phuong Le, Cell Metabolism Regenerative MedicineMedicineParathyroid HormoneParathyroid DiseaseStem Cell Research	2017	380
c-Ski Acts as a Transcriptional Co-repressor in Transforming Growth Factor-β Signaling through Interaction with Smads Shingo Akiyoshi, Hirofumi Inoue, Jun‐ichi Hanai, Journal of Biological Chemistry Cell ProliferationCell GrowthCellular PhysiologyTranscriptional RegulationSignaling Pathway	1999	376
A <i>RUNX2/PEBP2</i> αA/ <i>CBFA1</i> mutation displaying impaired transactivation and Smad interaction in cleidocranial dysplasia Yuwen Zhang, Natsuo Yasui, Kosei Ito, Proceedings of the National Academy of Sciences	2000	332
The muscle-specific ubiquitin ligase atrogin-1/MAFbx mediates statin-induced muscle toxicity Jun‐ichi Hanai, Peirang Cao, Preeti Tanksale, Journal of Clinical Investigation	2007	283
Endostatin Causes G1 Arrest of Endothelial Cells through Inhibition of Cyclin D1 Jun‐ichi Hanai, Mohan Dhanabal, S. Ananth Karumanchi, Journal of Biological Chemistry	2002	215
Hgs (Hrs), a FYVE Domain Protein, Is Involved in Smad Signaling through Cooperation with SARA Shigeto Miura, Toshikazu Takeshita, Hironobu Asao, Molecular and Cellular Biology	2000	176

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