Juliette Gafni - Concepedia

Concepedia

Author

Juliette Gafni

Buck Institute for Research on Aging

Also Known As

J Gafni, J. GAFNI, J. Gafni, Juliette Gafni

22

Publications

3.3K

Citations

19

H-Index

68

Concepts

All Affiliations

Buck Institute for Research on Aging University of California, San Francisco Johns Hopkins University CHDI Foundation Sanford Burnham Prebys Medical Discovery Institute

JG

About

Juliette Gafni is an author at Buck Institute for Research on Aging specializing in medicine, neurodegenerative diseases, and molecular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	14	2.5K
2	Natural Sciences	4	4	711
3	Social Sciences	2	2	438
4	Engineering	1	1	49
5	Health Sciences	1	1	57

Juliette Gafni

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19

Publications

3.3K

Citations

19

H-Index

	Year	Citations
Xestospongins: Potent Membrane Permeable Blockers of the Inositol 1,4,5-Trisphosphate Receptor Juliette Gafni, Julia A Munsch, Tien H. Lam, Neuron Proteinlipid InteractionSignal TransductionBiochemistryMedicineMembrane Transport	1997	591
Caspase Cleavage of Mutant Huntingtin Precedes Neurodegeneration in Huntington's Disease Cheryl L. Wellington, Lisa Ellerby, Claire‐Anne Gutekunst, Journal of Neuroscience GeneticsMolecular BiologyCaspase CleavageNeurochemical BiomarkersSynaptic Signaling	2002	398
Proteolysis of Mutant Huntingtin Produces an Exon 1 Fragment That Accumulates as an Aggregated Protein in Neuronal Nuclei in Huntington Disease Christian Landles, Kirupa Sathasivam, Andreas Weiss, Journal of Biological Chemistry	2010	333
Calpain Activation in Huntington's Disease Juliette Gafni, Lisa Ellerby Journal of Neuroscience Calpain CleavageMolecular BiologyNeurochemical BiomarkersCytoskeletonCalpain Activation	2002	324
Inhibition of Calpain Cleavage of Huntingtin Reduces Toxicity Juliette Gafni, Evan Hermel, Jessica E. Young, Journal of Biological Chemistry Calpain CleavageCell DeathCellular PhysiologyHd Tissue CultureToxicological Mechanism	2004	284
Specific caspase interactions and amplification are involved in selective neuronal vulnerability in Huntington's disease Evan Hermel, Juliette Gafni, Stephanie Propp, Cell Death and Differentiation Specific Caspase InteractionsSelective Neuronal VulnerabilityNeurochemical BiomarkersNeurologyNeuroscience	2004	221
FGF-2 promotes neurogenesis and neuroprotection and prolongs survival in a transgenic mouse model of Huntington's disease Kunlin Jin, Michelle LaFevre-Bernt, Yunjuan Sun, Proceedings of the National Academy of Sciences	2005	213
Matrix Metalloproteinases Are Modifiers of Huntingtin Proteolysis and Toxicity in Huntington's Disease John P. Miller, Jennifer Holcomb, Ismael Al‐Ramahi, Neuron BiochemistryMedicineNatural SciencesMatrix MetalloproteinasesMetalloprotein	2010	174
Huntingtin Phosphorylation Sites Mapped by Mass Spectrometry Birgit Schilling, Juliette Gafni, Cameron Torcassi, Journal of Biological Chemistry Molecular BiologyProtein ExpressionBioanalysisHuntington Disease PathogenesisDegenerative Pathology	2006	159
Brain infarction is not reduced in SOD-1 transgenic mice after a permanent focal cerebral ischemia Pak H. Chan, Hideyuki Kamii, Guo‐Yuan Yang, Neuroreport	1993	127

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