Christopher M. Counter

Author

Also Known As

C M Counter, C. Counter, C. M. Counter, C.M. Counter, CHRISTOPHER M. COUNTER, CM COUNTER, CM Counter, Chaikuad, Apirat, Chistopher M. Counter, Chris Counter

203

Publications

20.9K

Citations

H-Index

Concepts

All Affiliations

Duke Medical Center Duke University Hospital Duke University University of North Carolina at Chapel Hill Whitehead Institute for Biomedical Research

About

Christopher M. Counter is an author at Duke Medical Center specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	36	51	14.5K
2	Natural Sciences	5	5	331
3	Health Sciences	4	4	1.3K
4	Engineering	2	2	409
5	Biomedical Engineering	2	2	409

Christopher M. Counter

Publications

17.8K

Citations

H-Index

Page 1

	Year	Citations
Creation of human tumour cells with defined genetic elements William C. Hahn, Christopher M. Counter, Ante S. Lundberg, Nature Tumor BiologyHuman CellTumor HeterogeneityMedicineDefined Genetic Elements	1999	2.3K
Telomere shortening associated with chromosome instability is arrested in immortal cells which express telomerase activity. Christopher M. Counter, Ariel A. Avilion, Catherine LeFeuvre, The EMBO Journal ChromatinGenome InstabilityChromosome InstabilityDna DamageImmortal Cells	1992	2.1K
hEST2, the Putative Human Telomerase Catalytic Subunit Gene, Is Up-Regulated in Tumor Cells and during Immortalization Matthew Meyerson, Christopher M. Counter, Elinor Ng Eaton, Cell ChromatinCell RegulationOncogenic AgentMedicineMolecular Biology	1997	1.8K
A signalling pathway controlling c-Myc degradation that impacts oncogenic transformation of human cells Elizabeth S. Yeh, Melissa Cunningham, Hugh Arnold, Nature Cell Biology Signal TransductionCell RegulationC-myc DegradationHuman CellsOncogenic Agent	2004	783
Erk2 Phosphorylation of Drp1 Promotes Mitochondrial Fission and MAPK-Driven Tumor Growth Jennifer A. Kashatus, Aldo Nascimento, Lindsey J. Myers, Molecular Cell Tumor BiologyMitophagyMitochondrial FunctionErk2 PhosphorylationSystems Biology	2015	683
Dissociation among <i>in vitro</i> telomerase activity, telomere maintenance, and cellular immortalization Christopher M. Counter, William C. Hahn, Wenyi Wei, Proceedings of the National Academy of Sciences	1998	658
Copper is required for oncogenic BRAF signalling and tumorigenesis Donita C. Brady, Matthew S. Crowe, Michelle L. Turski, Nature Oncogenic AgentMetalloproteinMedicineCell BiologyTumor Biology	2014	581
The telomere hypothesis of cellular aging Calvin B. Harley, Homayoun Vaziri, Christopher M. Counter, Experimental Gerontology Lifespan ExtensionAgingCell DivisionLongevityTelomere Hypothesis	1992	489
Telomerase activity is restored in human cells by ectopic expression of hTERT (hEST2), the catalytic subunit of telomerase Christopher M. Counter, Matthew Meyerson, Elinor Ng Eaton, Oncogene ChromatinEctopic ExpressionTelomerase ActivityMolecular BiologyCatalytic Subunit	1998	422
Oncogenic Ras-induced secretion of IL6 is required for tumorigenesis Brooke Ancrile, Kian‐Huat Lim, Christopher M. Counter Genes & Development Cancer ResearchActive Oncogenic StateOncogenic AgentMedicineTumor Growth	2007	415

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