Thomas R. Bauer - Concepedia

Author

Thomas R. Bauer

National Cancer Institute ORCID

Also Known As

T Bauer, T R Bauer, T. Bauer, T. R. Bauer, T.R. Bauer, Thomas Bauer, Thomas R Bauer, Thomas R. Bauer, Thomas Roy Bauer

Publications

1.6K

Citations

H-Index

Concepts

All Affiliations

National Cancer Institute National Institutes of Health Center for Cancer Research Office of Research Services University of Washington

About

Thomas R. Bauer is an author at National Cancer Institute specializing in medicine, immunology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	19	1.1K
2	Health Sciences	2	2	214
3	Education	1	1	26
4	Engineering	1	1	12
5	Language Studies	1	1	12

Thomas R. Bauer

Publications

1.5K

Citations

H-Index

Page 1

	Year	Citations
Gains of glycosylation comprise an unexpectedly large group of pathogenic mutations Guillaume Vogt, Ariane Chapgier, Kun Yang, Nature Genetics Glycosylation CompriseBiosynthesisSomatic VariantLarge GroupPathogenesis	2005	209
Hypoxia regulatory elements of the human vascular endothelial growth factor gene. Susana Alicia Salceda, Thomas R. Bauer PubMed Human GrowthRedox BiologyOxidative StressAngiogenesisRedox Regulator	1994	199
Successful treatment of canine leukocyte adhesion deficiency by foamy virus vectors Thomas R. Bauer, James M. Allen, Mehreen Hai, Nature Medicine Veterinary VaccineSuccessful TreatmentFoamy Virus VectorsPathogenesisViral Pathogenesis	2007	153
The ets Family Member Tel Binds to the Fli-1 Oncoprotein and Inhibits Its Transcriptional Activity Boguslaw A. Kwiatkowski, L. Scot Bastian, Thomas R. Bauer, Journal of Biological Chemistry Mixed-phenotype Acute LeukemiaGeneticsMolecular BiologyMyeloid NeoplasiaHematological Malignancy	1998	129
A Missense Mutation in the β-2 Integrin Gene (ITGB2) Causes Canine Leukocyte Adhesion Deficiency James Kijas, Thomas R. Bauer, S. Gäfvert, Genomics β-2 Integrin GeneImmunogeneticsMissense MutationPathogenesisImmunology	1999	87
Haploidentical In Utero Hematopoietic Cell Transplantation Improves Phenotype and Can Induce Tolerance for Postnatal Same-Donor Transplants in the Canine Leukocyte Adhesion Deficiency Model William H. Peranteau, Todd E. Heaton, Yuchen Gu, Biology of Blood and Marrow Transplantation	2009	47
<i>ASXL1</i> and <i>STAG2</i> are common mutations in GATA2 deficiency patients with bone marrow disease and myelodysplastic syndrome Robert R. West, Katherine R. Calvo, Lisa J. Embree, Blood Advances Bone Marrow DiseaseDisease ProgressionGeneticsGenetic EpidemiologyPathology	2021	45
The human lambda L chain Ig locus. Recharacterization of JC lambda 6 and identification of a functional JC lambda 7 Thomas R. Bauer, Bonnie B. Blomberg The Journal of Immunology	1991	43
Correction of the disease phenotype in canine leukocyte adhesion deficiency using ex vivo hematopoietic stem cell gene therapy Thomas R. Bauer, Mehreen Hai, Laura M. Tuschong, Blood	2006	43
Long-Term Follow-up of Foamy Viral Vector-Mediated Gene Therapy for Canine Leukocyte Adhesion Deficiency Thomas R. Bauer, Laura M. Tuschong, Katherine R. Calvo, Molecular Therapy	2013	39

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