Juan Manuel Ramiro‐Diaz

Author

Also Known As

J. M. Ramiro-Díaz, J.M. Ramiro-Diaz, Juan M Ramiro‐Diaz, Juan M. Ramiro-Diaz, Juan Manuel Ramiro-Diaz, Juan Manuel Ramiro‐Diaz, Juan Ramiro-Diaz, Juan Ramiro-Díaz, Juan Ramiro‐Diaz, Juan Ramiro‐Díaz

Publications

226

Citations

H-Index

Concepts

All Affiliations

University of New Mexico

About

Juan Manuel Ramiro‐Diaz is an author at University of New Mexico specializing in medicine, cell biology, and vascular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	183
2	Cell Biology	4	4	147
3	Public Health	1	1	42
4	Biochemistry	1	1	42
5	Cellular Physiology	1	1	37

Juan Manuel Ramiro‐Diaz

Publications

198

Citations

H-Index

Page 1

	Year	Citations
Extracellular matrix 1 (ECM1) regulates the actin cytoskeletal architecture of aggressive breast cancer cells in part via S100A4 and Rho-family GTPases PC Gomez-Contreras, Juan Manuel Ramiro‐Diaz, Ana Sierra, Clinical & Experimental Metastasis Actin Cytoskeletal ArchitectureCell RegulationRho-family GtpasesMedicineCell Interaction	2016	49
NFAT is required for spontaneous pulmonary hypertension in superoxide dismutase 1 knockout mice Juan Manuel Ramiro‐Diaz, Carlos H. Nitta, Levi D. Maston, American Journal of Physiology-Lung Cellular and Molecular Physiology Lung InflammationPulmonary HypertensionRedox BiologyOxidative StressInflammation	2013	42
Interleukin‐6 trans‐signaling contributes to chronic hypoxia‐induced pulmonary hypertension Levi D. Maston, David T. Jones, Wieslawa Giermakowska, Pulmonary Circulation Lung InflammationImmunologyCellular PhysiologyInflammationImmunopathology	2018	37
Intermittent hypoxia-induced increases in reactive oxygen species activate NFATc3 increasing endothelin-1 vasoconstrictor reactivity Jessica Friedman, Carlos H. Nitta, K. M. Henderson, Vascular Pharmacology InflammationReactive Oxygen SpeciesMedicinePhysiologyHypoxia (Medicine)	2013	36
NFATc3 and VIP in Idiopathic Pulmonary Fibrosis and Chronic Obstructive Pulmonary Disease Anthony M. Szema, Edward Forsyth, Benjamin Ying, PLoS ONE Antifibrotic TherapyInflammatory Lung DiseaseAdvanced Lung DiseaseLung InflammationImmunology	2017	19
Luminal endothelial lectins with affinity for<i>N</i>-acetylglucosamine determine flow-induced cardiac and vascular paracrine-dependent responses Juan Manuel Ramiro‐Diaz, Alma Barajas‐Espinosa, Érika Chi-Ahumada, American Journal of Physiology-Heart and Circulatory Physiology	2010	15

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